Pancreatic cancer may evade the immune system not by accident but by actively switching off internal danger signals through ...

An international research team has succeeded in deciphering a key mechanism that controls the growth of pancreatic cancers.

Pancreatic cancer may evade the immune system using a clever molecular trick. Researchers found that the cancer-driving ...

Researchers say the findings offer a promising way to improve existing cancer therapies as well as develop new ones ...

Pancreatic cancer is notoriously hard to treat, often resisting therapies that target its most common mutations. Researchers have now uncovered a hidden three-part loop that fuels tumor growth, ...

MYC‑driven tumors silence innate immune alarms by clearing R‑loop–derived signals, exposing a new therapeutic angle for targeting MYC without broad inhibition.

Could this mark a shift in how we think about cancer therapy? At least in the laboratory, evidence suggests it may be . An ...

Please provide your email address to receive an email when new articles are posted on . Inhibiting RMB42 disrupted production of Myc proteins in pancreatic cancer cells. This approach could curtail ...

The growth protein, MYC, was consistently found with RBM42, which spurs MYC production, in human pancreatic tumors. Above, microscopy for MYC (red) and RBM42 (green) in a representative pancreatic ...

For decades, scientists have tried to stop cancer by disabling the mutated proteins that are found in tumors. But many cancers manage to overcome this and continue growing. Now, UCSF scientists think ...